{"university":{"openalex_id":"I146416000","name":"University of Kansas"},"topic":"age-crosslink-breakers","total_matching":3,"offset":0,"next_offset":null,"records":[{"doi":"https://doi.org/10.1161/hypertensionaha.112.195511","title":"Hypertension Induces Brain β-Amyloid Accumulation, Cognitive Impairment, and Memory Deterioration Through Activation of Receptor for Advanced Glycation End Products in Brain Vasculature","source_url":"https://doi.org/10.1161/hypertensionaha.112.195511","source_name":"Hypertension","cited_by_count":242,"is_open_access":true,"openalex_work_id":"W2119989228","publication_date":"2012-05-22","publication_year":2012,"university_research_work_topics":[{"topic_slug":"age-crosslink-breakers"}]},{"doi":"https://doi.org/10.1101/2024.05.14.594173","title":"Amyloid Beta Glycation Induces Neuronal Mitochondrial Dysfunction and Alzheimer’s Pathogenesis via VDAC1-Dependent mtDNA Efflux","source_url":"https://doi.org/10.1101/2024.05.14.594173","source_name":"bioRxiv (Cold Spring Harbor Laboratory)","cited_by_count":1,"is_open_access":true,"openalex_work_id":"W4396935461","publication_date":"2024-05-14","publication_year":2024,"university_research_work_topics":[{"topic_slug":"age-crosslink-breakers"}]},{"doi":"https://doi.org/10.1073/pnas.2505046122","title":"Amyloid-beta glycation induces neuronal mitochondrial dysfunction and Alzheimer’s pathogenesis via VDAC1-dependent mtDNA efflux","source_url":"https://doi.org/10.1073/pnas.2505046122","source_name":"Proceedings of the National Academy of Sciences","cited_by_count":8,"is_open_access":true,"openalex_work_id":"W4416293952","publication_date":"2025-11-17","publication_year":2025,"university_research_work_topics":[{"topic_slug":"age-crosslink-breakers"}]}],"automation_disclosure":"Generated automatically from cited source metadata. No scientist, clinician, researcher, editor, or human reviewer evaluates this publication before release."}