Butyrate
A familiar starting point. A wider world of research.
Follow butyrate through source-linked records, connected to Senolytics and Microbiome and dysbiosis. Explore the connections, then open the original evidence.
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A selection of other objects mentioned in these records. Inspect the shared source to see the connection; a shared mention does not mean the same effect.
sodium
Inspect supporting records 11dietary fiber
Inspect supporting records 2Sarcopenia
Inspect supporting records 2Autophagy
Inspect supporting records 1body weight
Inspect supporting records 1magnesium
Inspect supporting records 1More connections in this selection
Neuroinflammation
Inspect supporting records 1Oxidative stress
Inspect supporting records 1prebiotics
Inspect supporting records 1propionate
Inspect supporting records 1resistant starch
Inspect supporting records 1short chain fatty acids
Inspect supporting records 1soluble fiber
Inspect supporting records 1wheat
Inspect supporting records 1wheat bran
Inspect supporting records 1Where the research leads.
Published topic links attached to the selected records. Each dossier opens a wider field, including its own research ecosystem.
- SenolyticsRecords in this selection 12
- Microbiome and dysbiosisRecords in this selection 6
- Epigenetic alterationsRecords in this selection 5
- Centenarian biologyRecords in this selection 1
- Chronic inflammationRecords in this selection 1
- Magnesium and ageingRecords in this selection 1
- MetforminRecords in this selection 1
- NeuroinflammationRecords in this selection 1
- RapamycinRecords in this selection 1
- Regenerative medicineRecords in this selection 1
- SarcopeniaRecords in this selection 1
- Senescence-associated secretory phenotypeRecords in this selection 1
- Skin ageingRecords in this selection 1
- Vision ageingRecords in this selection 1
Meet the wider research ecosystem.
Follow a connected field into its universities, funding and patent activity.
University, grant and patent activity in a topic dossier concerns that wider field; it is not a butyrate-specific total.
Different records. Different questions.
This is the composition of the initial 24-record selection, not the entire collection. Open a group to inspect its supporting records. Publication types and existing automated evidence labels describe records, not their quality or conclusions. Unclassified records remain in “Other research records”.
The recent trail.
Recent sources in this selection: publication months for publications, registry update months for registrations.
Good questions to bring along.
What can I learn about butyrate here?
See which permitted records mention this object, follow the topics and other objects connected through those records, and inspect the sources. Whole foods, preparations, extracts and isolated compounds keep separate identities.
Do these records establish a benefit?
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Open a connected object and compare the actual records, preparations and research settings. “Mentioned in the same record” explains a navigation link; it does not establish equivalence, composition or causality.
How complete is this picture?
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Open the evidence.
Showing the records supporting this selection. Browse the full selection
Sodium butyrate attenuates D-galactose-induced cellular senescence in WI-38 fibroblasts in vitro via modulation of HDAC activity, Nrf2/ARE pathway, and p53/p21/p16 signaling.
Sodium butyrate-loaded nanoparticles coated with chitosan for the treatment of neovascularization in age-related macular degeneration: Ocular biocompatibility and antiangiogenic activity.
[Antiapoptotic gene bcl-2 prevents cellular senescence program reactivation induced by histone deacetylase inhibitor sodium butyrate in E1A and cHa-ras transformed rat fibroblasts].
[Involvement of MAP-kinase cascades into the regulation of sodium butyrate induced premature cell senescence].
SRSF2 is required for sodium butyrate-mediated p21(WAF1) induction and premature senescence in human lung carcinoma cell lines.
Posttraining systemic administration of the histone deacetylase inhibitor sodium butyrate ameliorates aging-related memory decline in rats.
[Cell senescence induced by histone deacetylase inhibitor sodium butyrate in rodent transformed cells resistant to apoptosis].
p21(Waf1) is required for cellular senescence but not for cell cycle arrest induced by the HDAC inhibitor sodium butyrate.
[Sodium butyrate do not induce the program of premature senescence in transformants with JNK1,2 knockout].
[Induction of premature senescence program by an inhibitor of histone deacetylase sodium butyrate in normal and transformed rat fibroblasts].
Sodium butyrate induces growth arrest and senescence-like phenotypes in gynecologic cancer cells.
About this collection: scope and limitations
Nutrient or compound · Compound identity, formulation and the studied outcome matter. A record about a related substance, a product or a mechanism does not automatically apply to this identity.
Questions to check in the original studies
- Which compound and formulation does the source identify?
- Which population and outcome were studied?
- Is an observation about a measured outcome being distinguished from a claim about ageing or lifespan?
What the displayed selection contains
24 research metadata records and 0 trial registrations were returned in the initial selection. More records can be loaded above. This is not a complete worldwide evidence review. These counts do not measure benefit, safety or evidence certainty.
Interpretation remains unassessed
Open the original sources to assess study design, findings and applicability. A trial registration is not a result; a metadata mention is not a scientific conclusion. Missing matches do not establish that research is absent.
Index snapshot: . This is the index projection timestamp, not the date of a discovery or expert review.
Cite this research-navigation view
immortal.life. butyrate: research mentions. https://www.immortal.life/explore/butyrate. Projection snapshot: 2026-10-16T00:50:00.236Z.
Include your access date. This citation identifies an automated navigation view, not a clinical conclusion. The live selection can change with corrections and source permissions.
Matching, coverage and vocabulary provenance
This automated index uses permitted minimal bibliographic metadata and checks current source and record eligibility. Literal title or controlled-term matches are discovery relationships, not scientific findings. Retained metadata coverage is bounded; historical completeness of external sources is not certified.
Foods, preparations, extracts and compounds retain distinct identities. Ambiguous labels require a choice. Source AI processing, model training and raw redistribution are disabled for this search.
Vocabulary attribution: FoodOn contributors, CC BY 4.0, filtered own-namespace labels/IDs and references; courtesy of the U.S. National Library of Medicine, 2026 MeSH vocabulary terms, filtered labels/entry terms; USDA FoodData Central, Foundation Foods April 2026, CC0, filtered identifiers/descriptions. No source endorsement. Navigation spelling/plural changes are separately attributed.